Aspirin Inhibits Chlamydia pneumoniae–Induced Nuclear Factor- B Activation, Cytokine Expression, and Bacterial Development in Human Endothelial Cells

نویسندگان

  • Beate Tiran
  • Andreas Tiran
  • Hans-Jürgen Gruber
  • Wolfgang F. Graier
  • Andreas H. Wagner
  • Ellen B.M. van Leeuwen
چکیده

Andreas Tiran, Hans-Jürgen Gruber, Wolfgang F. Graier, Andreas H. Wagner, Ellen B.M. van Expression, and Bacterial Development in Human Endothelial Cells B Activation, Cytokine κ Induced Nuclear Factor− Chlamydia pneumoniae Aspirin Inhibits Print ISSN: 1079-5642. Online ISSN: 1524-4636 Copyright © 2002 American Heart Association, Inc. All rights reserved. Greenville Avenue, Dallas, TX 75231 is published by the American Heart Association, 7272 Arteriosclerosis, Thrombosis, and Vascular Biology doi: 10.1161/01.ATV.0000022695.22369.BE 2002;22:1075-1080; originally published online May 23, 2002; Arterioscler Thromb Vasc Biol. http://atvb.ahajournals.org/content/22/7/1075 World Wide Web at: The online version of this article, along with updated information and services, is located on the http://atvb.ahajournals.org/content/suppl/2002/07/15/22.7.1075.DC1.html Data Supplement (unedited) at:

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Aspirin inhibits Chlamydia pneumoniae-induced nuclear factor-kappa B activation, cytokine expression, and bacterial development in human endothelial cells.

OBJECTIVE Chlamydia pneumoniae has been associated with atherosclerosis. Infection of vascular endothelial cells with C pneumoniae increases the expression of proatherogenic cytokines mediated by nuclear factor (NF)-kappaB, a transcription factor. The present study was designed to test the effect of aspirin on C pneumoniae-induced NF-kappaB activation, interleukin expression, and bacterial deve...

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Aspirin Inhibits Chlamydia pneumoniae–Induced Nuclear Factor-kB Activation, Cytokine Expression, and Bacterial Development in Human Endothelial Cells

Objective—Chlamydia pneumoniae has been associated with atherosclerosis. Infection of vascular endothelial cells with C pneumoniae increases the expression of proatherogenic cytokines mediated by nuclear factor (NF)-kB, a transcription factor. The present study was designed to test the effect of aspirin on C pneumoniae–induced NF-kB activation, interleukin expression, and bacterial development ...

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تاریخ انتشار 2002